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محتوای ارائه شده توسط AgingYounger. تمام محتوای پادکست شامل قسمت‌ها، گرافیک‌ها و توضیحات پادکست مستقیماً توسط AgingYounger یا شریک پلتفرم پادکست آن‌ها آپلود و ارائه می‌شوند. اگر فکر می‌کنید شخصی بدون اجازه شما از اثر دارای حق نسخه‌برداری شما استفاده می‌کند، می‌توانید روندی که در اینجا شرح داده شده است را دنبال کنید.https://fa.player.fm/legal
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Telomere Shorting is how our bodies age-Lengthen Them & Live Longer

31:00
 
اشتراک گذاری
 

Manage episode 235999435 series 1024467
محتوای ارائه شده توسط AgingYounger. تمام محتوای پادکست شامل قسمت‌ها، گرافیک‌ها و توضیحات پادکست مستقیماً توسط AgingYounger یا شریک پلتفرم پادکست آن‌ها آپلود و ارائه می‌شوند. اگر فکر می‌کنید شخصی بدون اجازه شما از اثر دارای حق نسخه‌برداری شما استفاده می‌کند، می‌توانید روندی که در اینجا شرح داده شده است را دنبال کنید.https://fa.player.fm/legal
Telomeres function similarly, preventing chromosomes from fraying or tangling with one another. When that happens, it can cause genetic information to get mixed up or destroyed, leading to cell malfunction, increasing the risk of disease or even shortening lifespan. Each time a cell divides, its telomeres become shorter. After years of splicing and dicing, telomeres become too short for more divisions. At this point, cells are unable to divide further and become inactive, or die. As more of our cells lose their telomeres and go out of commission, without others to take their place, the body follows and begins breaking down. Telomeres don’t leave (or shorten) quietly. Their shortening process has been linked with aging, cancer and a higher risk of death. A study found that individuals with the shortest telomeres, or about 10 percent of the study’s participants, were 23 percent more likely to die within three years than those with longer telomeres. There is an enzyme called Telomerase that lengthens telomeres and keeps them from wearing out too fast or too early, which helps you live longer. By reawakening telomerase in human cells where it’s stopped working, normal human aging could be slowed. Want to start Aging Younger? Tune in Wednesday JUNE 12TH at 7PM FOR THE REST OF THE STORY! http://tobtr.com/s/11375979
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301 قسمت

Artwork
iconاشتراک گذاری
 
Manage episode 235999435 series 1024467
محتوای ارائه شده توسط AgingYounger. تمام محتوای پادکست شامل قسمت‌ها، گرافیک‌ها و توضیحات پادکست مستقیماً توسط AgingYounger یا شریک پلتفرم پادکست آن‌ها آپلود و ارائه می‌شوند. اگر فکر می‌کنید شخصی بدون اجازه شما از اثر دارای حق نسخه‌برداری شما استفاده می‌کند، می‌توانید روندی که در اینجا شرح داده شده است را دنبال کنید.https://fa.player.fm/legal
Telomeres function similarly, preventing chromosomes from fraying or tangling with one another. When that happens, it can cause genetic information to get mixed up or destroyed, leading to cell malfunction, increasing the risk of disease or even shortening lifespan. Each time a cell divides, its telomeres become shorter. After years of splicing and dicing, telomeres become too short for more divisions. At this point, cells are unable to divide further and become inactive, or die. As more of our cells lose their telomeres and go out of commission, without others to take their place, the body follows and begins breaking down. Telomeres don’t leave (or shorten) quietly. Their shortening process has been linked with aging, cancer and a higher risk of death. A study found that individuals with the shortest telomeres, or about 10 percent of the study’s participants, were 23 percent more likely to die within three years than those with longer telomeres. There is an enzyme called Telomerase that lengthens telomeres and keeps them from wearing out too fast or too early, which helps you live longer. By reawakening telomerase in human cells where it’s stopped working, normal human aging could be slowed. Want to start Aging Younger? Tune in Wednesday JUNE 12TH at 7PM FOR THE REST OF THE STORY! http://tobtr.com/s/11375979
  continue reading

301 قسمت

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